PROTOCOLS
Aflatoxin
ASPERGILLUS FLAVUS, A. PARASITICUS
Aflatoxins as a class are acutely toxic. They primarily impact the liver, however the detoxification burden is also shared with the kidneys.
Gliotoxin
ASPERGILLUS SPP., TRICHODERMA SPP.
Gliotoxin opens the doorway to fungal infection. It can be produced in vivo, contributing to the etiology of fungal infections, and is a virulence factor for the airborne pathogen Aspergillus fumigatus.
Mycophenolic Acid
PENICILLIUM SPP., ASPERGILLUS SPP., EUROTIUM REPENS
Mycophenolic acid (MPA) is a mold metabolite that’s excreted from mold as it’s digesting and metabolizing the substrate it’s growing upon.
Increased MPA is highly suggestive of current exposure to mold, because where there’s living mold, there’s MPA, but not necessarily mycotoxins.
Ochratoxin
ASPERGILLUS OCHRASEUS, A. NIGER, PENICILLIUMVERRUCOSUM, P. NORDICUM, P. CHRYSOGENUM
Ochratoxin is a persister mycotoxin. It binds tightly to the blood protein albumin, acting like a mobile reserve, resulting in an extended time in the blood. The kidneys are the primary route of excretion, which sustain significant damage in the process.
Mold & Mycotoxins
Mycotoxins are secondary metabolites that are released from mold as a competitive
advantage over other microbes in a hospitable environment, which is related to spaces with
higher levels of humidity. Many mold species that also enjoy an environment as humans do
are mycotoxin formers.